[1] Jansen JP, Fleurence R, Devine B, et al. Interpreting Indirect Treatment Comparisons and Network Meta-Analysis for Health-Care Decision Making: Report of the ISPOR Task Force on Indirect Treatment Comparisons Good Research Practices: Part 1[J]. Value in Health, 2011, 14(4):417-428. [2] Lu G, Ades AE. Combination of direct and indirect evidence in mixed treatment comparisons[J]. Statistics in Medicine, 2004, 23(20):3105-3124. [3] American Cancer Society. Cancer Facts and Figures 2014. Atlanta, Ga: American Cancer Society, 2014.SAvailable online. Last accessed October 2, 2014. [4] Bayraktar S,SGl?ck S.MolecularlyStargetedStherapiesSforSmetastaticStriple-negative breast cancer[J]. BreastSCancerSRes Treat,S2013,138(1):21-35. [5] Cheang MC, Voduc D, Bajdik C, et al. Basal-like breast cancer defined by five biomarkers has superior prognostic value than triple-negative phenotype[J]. Clin Cancer Res, 2008,14:1368-1376. [6] Bauer KR, Brown M, Cress RD, et al. Descriptive analysis of estrogen receptor (ER)-negative, progesterone receptor (PR)-negative, and HER2-negative invasive breast cancer, the so-called triple-negative phenotype: a population-based study from the California cancer Registry[J]. Cancer, 2007,109:1721-1728. [7] Cleator S,SHeller W,SCoombes RC.Triple-negative breast cancer:StherapeuticSoptions[J].Lancet Oncol,S2007,8(3):235-244. [8] Brower V. SearchSforSnewStreatmentsSintensifiesSforStriple-negativeSbreastScancer[J]. J NatlSCancerSInst, 2009;101(22):1536-1537. [9] Clark OAC, Engel T, Paladini L, et al. Targeted Therapy in Triple-Negative Metastatic Breast Cancer (TNBC) - A Systematic Review and Meta-Analysis[J]. Core Evid, 2013, 9(9):1-11. [10] Bramati A, Girelli S, Torri V, et al. Efficacy of biological agents in metastatic triple-negative breast cancer[J]. Cancer Treat Rev, 2014, 40(5):605-613. [11] Brufsky A,SValero V,STiangco B,Set al. Second-line bevacizumab-containing therapy in patients with triple-negative breast cancer: subgroup analysis of the RIBBON-2 trial[J].Breast Cancer Res Treat, 2012,133(3):1067-1075. [12] Brufsky AM,SHurvitz S,SPerez E,Set al. RIBBON-2: a randomized, double-blind, placebo-controlled, phase III trial evaluating the efficacy and safety of bevacizumab in combination with chemotherapy for second-line treatment of human epidermal growth factor receptor 2-negative metastatic breast cancer[J].J Clin Oncol, 2011,29(32):4286-4293. [13] O'Shaughnessy J, Osborne C, Pippen JE, et al.Iniparib plus chemotherapy in metastatic triple-negative breast cancer[J].N Engl J Med, 2011,364(3):205-2014. [14] Finn RS,SPress MF,SDering J,Set al. Estrogen receptor,Sprogesterone receptor,Shuman epidermal growth factor receptor 2S(HER2), andSepidermal growth factor receptorSexpressionSandSbenefitSfromSlapatinibSin aSrandomizedStrialSofSpaclitaxelSwithSlapatinibSor placeboSasSfirst-lineStreatmentSinSHER2-negativeSorSunknownSmetastaticSbreast cancer[J]. J Clin Oncol,S2009,27(24):3908-3915. [15] Curigliano G,SPivot X,SCort?s J,Set al.Randomized phase II study of sunitinib versus standard of care for patients with previously treated advanced triple-negative breast cancer[J].Breast, 2013,22(5):650-656. [16] Tr?dan O, Campone M, Jassem J,Set al.Ixabepilone Alone or With Cetuximab as First-Line Treatment for Advanced/Metastatic Triple-Negative Breast Cancer[J].Clin Breast Cancer, 2015,15(1):8-15. [17] Carey LA,SRugo HS,SMarcom PK,Set al.TBCRC 001: randomized phase II study of cetuximab in combination with carboplatin in stage IV triple-negative breast cancer[J].J Clin Oncol, 2012,30(21):2615-2623. [18] Baselga J,SG?mez P,SGreil R,Set al.Randomized phase II study of the anti-epidermal growth factor receptor monoclonal antibody cetuximab with cisplatin versus cisplatin alone in patients with metastatic triple-negative breast cancer[J].J Clin Oncol, 2013,31(20):2586-2592. [19] Baselga J,SSegalla JG,SRoch? H,Set al. Sorafenib in combination with capecitabine: an oral regimen for patients with HER2-negative locally advanced or metastatic breast cancer[J].J Clin Oncol, 2012,30(13):1484-1491. [20] Bergh J,SBondarenko IM,SLichinitser MR, et al. First-line treatment of advanced breast cancer with sunitinib in combination with docetaxel versus docetaxel alone: results of a prospective, randomized phase III study[J].J Clin Oncol, 2012,30(9):921-929. [21] Pivot X,SSchneeweiss A,SVerma S,Set al. Efficacy and safety of bevacizumab in combination with docetaxel for the first-line treatment of elderly patients with locally recurrent or metastatic breast cancer: results from AVADO[J]. Eur J Cancer, 2011, 47(16):2387-2395. [22] Miller K,SWang M,SGralow J,Set al.Paclitaxel plus bevacizumab versus paclitaxel alone for metastatic breast cancer[J].N Engl J Med, 2007,357(26):2666-2676. [23] Robert NJ, Di?ras V,SGlaspy J,Set al. RIBBON-1:Srandomized,Sdouble-blind,Splacebo-controlled,Sphase III trialSofSchemotherapySwith or without bevacizumabSforSfirst-lineStreatmentSofShumanSepidermal growth factor receptorS2-negative,SlocallySrecurrentSor metastaticSbreast cancer[J]. J Clin Oncol,S2011,29(10):1252-1260. [24] O'Shaughnessy J,SSchwartzberg L,SDanso MA, et al.Phase III study of iniparib plus gemcitabine and carboplatin versus gemcitabine and carboplatin in patients with metastatic triple-negative breast cancer[J].J Clin Oncol,2014,32(34):3840-3847. [25] 韩华正, 李永安. 三阴性乳腺癌分子靶向治疗的研究进展[J]. 临床肿瘤学杂志, 2012, 17(5):470-474.
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